Mosquitoes are one of my favorite venomous animals. These natural phlebotomists have efficient venoms which allow them to effortlessly violate our most precious tissue—our blood—while manipulating our immune system to remain under the radar. You can just imagine how hard that venom has to work to hide the invading mouthparts, which poke around in search of a suitable capillary, as this awesome video from KQED’s Deep Look shows:
You can tell a lot about an book by the author’s photo. My author photo for Venomous, for example, paints me as the intrepid explorer; I look adventurous and daring as I smile unabashedly through the legs of a large tailless whip scorpion (amblypygid). But while the photo is startling, it’s not as bold as it seems. It’s a facade of bravado, not real bravery, as the menacing-looking animal on my cheek is actually harmless. Meanwhile, in his author photo for Sting of the Wild, Justin Schmidt shows that he’s far more daring: on his nose crawls a large Dinoponera ant.
Schmidt probably would laugh at my assessment of the image; after all, he refers to the Dinoponera, the largest ants on Earth, as the “gentle giants of the ant world.” But I went with a non-stinging amblypigid rather than an ant for a reason. According to Schmidt’s own colorful and cheeky index detailing the pain of insect stings, the ant crawling across his face possesses “A pulsing sting with some flavor.” Sure, it only scores a 1.5 out of 4.0 on his pain scale—but that’s 1.5 more painful than any species I would place on my snout.
It’s a photo befitting the book of a man who has been stung by more than 1000 times by some 80+ different species. As an entomologist who studies the Hymenoptera—bees, wasps and ants—Schmidt has a lifetime of experiences to draw upon for Sting of the Wild, his first book, which which hit shelves May 15th. Read More
April marked the twelfth consecutive month of record-breaking temperatures. That’s an entire year of our planet, on land and in the sea, being hotter-than-ever-recorded since record keeping began in 1880. Such extraordinary warmth is affecting ecosystems globally, but perhaps the hardest hit are coral reefs, whose fundamental organisms are incredibly sensitive to the heat.
Earlier this year, authorities in Australia reported that the Great Barrier Reef was in the midst of its worst bleaching event ever. Surveys above and below the water estimated that over 90% of the reefs were affected by bleaching. Now, as the summer wanes down under, scientists are finally able to begin to assess the lasting damage caused by this event. Their findings are heartbreaking. Read More
The global problem of snakebite goes unnoticed by most of us in developed countries. We have good access to medical care, abundant antivenom to treat what few dangerous bites occur, and snake species whose venoms are often manageable. In the U.S., for example, bees and wasps kill over ten times as many people as snakes do. But in other countries, snakebite is a real and neglected problem. Worldwide, snakes claim more than one hundred thousands lives annually, and leave countless more disabled and disfigured. This powerful, heartbreaking seven and a half-minute video is one that everyone should see:
Rising carbon dioxide levels in our atmosphere are changing Earth’s climate at an unprecedented rate. Not only is our planet getting warmer on average—in the oceans, a chemical reaction spurred by dissolved CO2 is altering water chemistry, causing a decrease in pH. This effect of climate change, called ocean acidification, can dissolve the calcium carbonate foundations of coral reefs and other calcifying organisms, making it impossible to build and maintain healthy reefs. Luckily, recent studies on how corals react to lower pHs has given scientists hope that they may be more resilient than previously thought. However, to truly understand how reefs will respond to climate change, we have to look at more than just corals.
Reefs are complex ecosystems, the bases of which are comprised of so much more than corals. There are other species which act as calcifiers, adding to the carbonate foundation (such as crustose coralline algae). The contribution of these non-coral species to reef growth, called secondary accretion, helps shape the surface and guide the settlement of larval corals. There are also species that eat away at the reef, including many worms and sponges. These bioeroders can weaken reef structures until they crumble apart. Whether a reef grows or shrinks over time depends on the interplay between its corals, other reef-builders, and the burrowing organisms which eat their way through the reef’s carbonate foundation. Read More
When you look one of these little snakes in its adorable little face, it’s not hard to see how the hognose got its name. Their upturned snoots give the snakes a porcine appearance.
But hognoses don’t just have adorable nasal features—they are also the drama queens of the serpent world. If you thought William Shatner wins the prize for worst over-actor on the planet, think again:
The end in particular just slays me: “No, I’m dead. See? I’m dead. So dead. Belly up dead.” Read More
The moment a viper’s venom enters the body, its enzymatic components set about their nefarious work. Metalloproteases begin the assault by mowing down structurally essential components of blood vessels and tissues, weakening walls and making holes that leech fluid. Capillaries hemmorrhage and the area swells while the proteases keep at their attack, taking out skeletal muscle through mechanisms poorly understood. Phospholipases join in, with their sights set on cell membranes. Some cut apart membrane lipids making lethal holes, while others seem to be just as destructive without enzymatic activity. The end result: muscle tissue dies. Hyaluronidases and serine proteases aid in the efforts, and the helpless tissue succumbs to the venom’s siege.
And that’s not even the worst part. The metalloproteases and phospholipases have other tricks up their sleeves. They don’t just fight their own war on our flesh: they enlist our own immune system to help them do it. The liberation of tumor necrosis factor and immune-stimulating cytokines by metalloproteases and the release of bioactive lipids by phospholipases cause immune cells to rush to the wound. Our body’s forces are trained to kill, usually setting their sights on bacteria and viruses. But without those clear targets, the body’s army gets confused. They can’t tell friend from foe, yet the immune cells fire anyway, blindly attacking an unseen danger. Valiant volleys act as friendly fire, adding to the death toll of innocent tissues.
Though scientists have been warning about the disastrous impacts that climate change will have on our planet for decades, we are now starting to feel those predictions manifest. As Eric Holthaus pointed out, the “worst nightmare” scenarios are already happening. Droughts, storms, fires, you name it—the world as we knew it is under siege. Heck, we just had the most abnormally hot month on record; February 2016 was 1.35 degrees Celsius warmer than the average, making it two-tenths of a degree more unusually warm than the previous record month: January 2016.
And as water supplies dwindle, rainforests burn, and corals bleach, we may have yet another thing to worry about: frickin’ snakes.
Many know Neil deGrasse Tyson for his pithy, humorous science tweets, which are a part of his greater science communication strategy. As of late, though, scientists have become quite vexed with NDT’s 140-character stylings, as he’s been foraying outside his planetary expertise and into biological phenomena, getting the facts wrong every time. First, there was his mistaken evaluation of evolutionary drivers and how sex works, excellently torn apart by Emily Willingham (a Ph.D. scientist whom Tyson then condescendingly called “a woman who has a blog”, prompting some to suggest he be referred to as just a “man with a twitter”). Then came his misunderstanding of genetics and deleterious alleles, which was ripped apart by Jeremy Yoder (another Ph.D. scientist). Now, he’s stepped in guano again with this tweet related to this weekend’s powerhouse movie release:
If Batman wants so badly to be a bat, he might be more intriguing if (like Marvel’s Daredevil) he were also blind, like a Bat
— Neil deGrasse Tyson (@neiltyson) March 25, 2016
Alas, the phrase “blind as a bat” is simply wrong. Let me explain why:
It’s official: Lee Se-dol has lost his first two Go games against AlphaGo, the computer program from Google’s DeepMind. Going into the match, Lee said he was confident, predicting victory in all 5 games. So when he lost the first game, he was shellshocked: “I didn’t expect to lose,” he said.”Even when I was behind, I still didn’t imagine that I’d lose. I didn’t think that it would be able to play such an excellent game.”
He’s now 0-2 out of 5 against AlphaGo, with $1 million on the line.
As Lee sat in front of the press after the second loss, he looked visibly shaken. “Yesterday I was surprised but today it’s more than that — I am speechless,” he said. Lee rocked back and forth slightly while DeepMind founder Demis Hassabis described the program’s confidence through the game, fidgeting as the cameras snapped hundreds of photos. He has a day to think about his strategy before game 3 on Saturday
I can understand how for some, a person losing a board game to a computer might seem inconsequential; after all, the best minds in Chess were beaten by computers decades ago. But this isn’t Chess. Go, a roughly 3,000 year old game (called as weiqi in China, igo in Japan, and baduk in Korea), is staggeringly more complex than other strategy board games. It’s estimated that there are some 10761 possible games of Go (compared with 10120 for Chess)—more than the number of atoms in the known universe. This means that even the most powerful computers on the planet can’t calculate ahead to conclusively determine the best move to play. Human players rely on a mix of skill, instinct, and imagination.
I know quite well how much of a challenge it is to program a machine to mimic the art of play. After all, my dad wrote the first commercial Go program. Read More